{"product_id":"cardio-zoomer-plus-cardio-genetics","title":"Cardio Zoomer plus Cardio Genetics","description":"\u003cp\u003e\u003cbr\u003e\u003c\/p\u003e\n\u003cstyle\u003e\n.nhd { font-family: Georgia, serif; color: #1a0a0d; max-width: 860px; line-height: 1.75; }\n.nhd-hook { background: #722F37; color: #fff; padding: 2.25rem 2rem 2rem; border-radius: 8px; margin-bottom: 2rem; }\n.nhd-hook h2 { font-size: 1.6rem; font-weight: 700; margin: 0 0 .75rem; color: #fff; line-height: 1.3; }\n.nhd-hook p { margin: 0; font-size: 1rem; color: rgba(255,255,255,0.88); }\n.nhd-stat-bar { display: grid; grid-template-columns: repeat(3,1fr); gap: 12px; margin-bottom: 2rem; }\n.nhd-stat { background: #1a0a0d; color: #fff; border-radius: 8px; padding: 1.1rem 1rem; text-align: center; }\n.nhd-stat-num { font-size: 1.8rem; font-weight: 700; color: #C8973A; line-height: 1; margin-bottom: .3rem; }\n.nhd-stat-label { font-size: .75rem; color: rgba(255,255,255,.78); line-height: 1.4; }\n.nhd-lead { font-size: 1.05rem; 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}\n}\n\u003c\/style\u003e\n\u003cdiv class=\"nhd\"\u003e\n\u003cdiv class=\"nhd-hook\"\u003e\n\u003ch2\u003eEvery year, half of all first heart attacks happen to people whose cholesterol was \"normal.\"\u003c\/h2\u003e\n\u003cp\u003eStandard lipid testing was designed in an era before we understood inflammation, oxidative stress, genetics, or endothelial function. It was never designed to prevent heart attacks — and it shows. The Cardio Zoomer was built for one purpose: to find what's actually killing people before it does.\u003c\/p\u003e\n\u003c\/div\u003e\n\u003cdiv class=\"nhd-stat-bar\"\u003e\n\u003cdiv class=\"nhd-stat\"\u003e\n\u003cdiv class=\"nhd-stat-num\"\u003e50%\u003c\/div\u003e\n\u003cdiv class=\"nhd-stat-label\"\u003eof heart attack victims had \"normal\" LDL cholesterol\u003c\/div\u003e\n\u003c\/div\u003e\n\u003cdiv class=\"nhd-stat\"\u003e\n\u003cdiv class=\"nhd-stat-num\"\u003e200+\u003c\/div\u003e\n\u003cdiv class=\"nhd-stat-label\"\u003ebiomarkers and genetic variants assessed in one panel\u003c\/div\u003e\n\u003c\/div\u003e\n\u003cdiv class=\"nhd-stat\"\u003e\n\u003cdiv class=\"nhd-stat-num\"\u003e#1\u003c\/div\u003e\n\u003cdiv class=\"nhd-stat-label\"\u003ecause of death in America — still. Heart disease kills one person every 33 seconds.\u003c\/div\u003e\n\u003c\/div\u003e\n\u003c\/div\u003e\n\u003cp class=\"nhd-lead\"\u003eThe tests your doctor orders were designed to be fast, cheap, and easy to bill — not to save your life. The Cardio Zoomer was designed by scientists who asked a different question: \u003cem\u003ewhat does it actually take to see cardiovascular risk before it becomes catastrophic?\u003c\/em\u003e The answer is 200+ markers. Here's why nothing else comes close.\u003c\/p\u003e\n\u003cdiv class=\"nhd-section\"\u003e\n\u003cdiv class=\"nhd-h3\"\u003eWhy every other test is leaving you exposed\u003c\/div\u003e\n\u003cp\u003eBefore we tell you what the Cardio Zoomer does, you need to understand what the tests you've already had are missing — and why that gap could cost you your life.\u003c\/p\u003e\n\u003c\/div\u003e\n\u003cdiv class=\"nhd-compare\"\u003e\n\u003cdiv class=\"nhd-compare-head\"\u003e\n\u003cdiv\u003eWhat it measures\u003c\/div\u003e\n\u003cdiv style=\"text-align: center;\"\u003eStandard tests\u003c\/div\u003e\n\u003cdiv style=\"text-align: center;\"\u003eCardio Zoomer\u003c\/div\u003e\n\u003c\/div\u003e\n\u003cdiv class=\"nhd-compare-row\"\u003e\n\u003cdiv\u003eBasic cholesterol (LDL, HDL, TG)\u003c\/div\u003e\n\u003cdiv class=\"chk\" style=\"text-align: center;\"\u003eYes\u003c\/div\u003e\n\u003cdiv class=\"chk\" style=\"text-align: center;\"\u003eYes\u003c\/div\u003e\n\u003c\/div\u003e\n\u003cdiv class=\"nhd-compare-row\"\u003e\n\u003cdiv\u003eApoB (true atherogenic particle count)\u003c\/div\u003e\n\u003cdiv class=\"x\" style=\"text-align: center;\"\u003eRarely\u003c\/div\u003e\n\u003cdiv class=\"chk\" style=\"text-align: center;\"\u003eYes\u003c\/div\u003e\n\u003c\/div\u003e\n\u003cdiv class=\"nhd-compare-row\"\u003e\n\u003cdiv\u003eLp(a) — inherited heart attack risk\u003c\/div\u003e\n\u003cdiv class=\"x\" style=\"text-align: center;\"\u003eAlmost never\u003c\/div\u003e\n\u003cdiv class=\"chk\" style=\"text-align: center;\"\u003eYes\u003c\/div\u003e\n\u003c\/div\u003e\n\u003cdiv class=\"nhd-compare-row\"\u003e\n\u003cdiv\u003eSmall dense LDL (the dangerous particles)\u003c\/div\u003e\n\u003cdiv class=\"x\" style=\"text-align: center;\"\u003eNo\u003c\/div\u003e\n\u003cdiv class=\"chk\" style=\"text-align: center;\"\u003eYes\u003c\/div\u003e\n\u003c\/div\u003e\n\u003cdiv class=\"nhd-compare-row\"\u003e\n\u003cdiv\u003eCeramides (plaque-forming fat signals)\u003c\/div\u003e\n\u003cdiv class=\"x\" style=\"text-align: center;\"\u003eNo\u003c\/div\u003e\n\u003cdiv class=\"chk\" style=\"text-align: center;\"\u003eYes\u003c\/div\u003e\n\u003c\/div\u003e\n\u003cdiv class=\"nhd-compare-row\"\u003e\n\u003cdiv\u003eOxidized LDL (LDL that's actually dangerous)\u003c\/div\u003e\n\u003cdiv class=\"x\" style=\"text-align: center;\"\u003eNo\u003c\/div\u003e\n\u003cdiv class=\"chk\" style=\"text-align: center;\"\u003eYes\u003c\/div\u003e\n\u003c\/div\u003e\n\u003cdiv class=\"nhd-compare-row\"\u003e\n\u003cdiv\u003eInflammation (hsCRP, IL-6, TNF-α)\u003c\/div\u003e\n\u003cdiv class=\"part\" style=\"text-align: center;\"\u003eSometimes CRP\u003c\/div\u003e\n\u003cdiv class=\"chk\" style=\"text-align: center;\"\u003eAll 6\u003c\/div\u003e\n\u003c\/div\u003e\n\u003cdiv class=\"nhd-compare-row\"\u003e\n\u003cdiv\u003eInsulin resistance \/ metabolic drivers\u003c\/div\u003e\n\u003cdiv class=\"x\" style=\"text-align: center;\"\u003eRarely\u003c\/div\u003e\n\u003cdiv class=\"chk\" style=\"text-align: center;\"\u003e30 markers\u003c\/div\u003e\n\u003c\/div\u003e\n\u003cdiv class=\"nhd-compare-row\"\u003e\n\u003cdiv\u003eEndothelial function (ADMA, nitric oxide)\u003c\/div\u003e\n\u003cdiv class=\"x\" style=\"text-align: center;\"\u003eNever\u003c\/div\u003e\n\u003cdiv class=\"chk\" style=\"text-align: center;\"\u003eYes\u003c\/div\u003e\n\u003c\/div\u003e\n\u003cdiv class=\"nhd-compare-row\"\u003e\n\u003cdiv\u003eOxidative stress (cell and vessel damage)\u003c\/div\u003e\n\u003cdiv class=\"x\" style=\"text-align: center;\"\u003eNever\u003c\/div\u003e\n\u003cdiv class=\"chk\" style=\"text-align: center;\"\u003eYes\u003c\/div\u003e\n\u003c\/div\u003e\n\u003cdiv class=\"nhd-compare-row\"\u003e\n\u003cdiv\u003eGenetic cardiovascular risk variants\u003c\/div\u003e\n\u003cdiv class=\"x\" style=\"text-align: center;\"\u003eNever\u003c\/div\u003e\n\u003cdiv class=\"chk\" style=\"text-align: center;\"\u003e120+ variants\u003c\/div\u003e\n\u003c\/div\u003e\n\u003cdiv class=\"nhd-compare-row\"\u003e\n\u003cdiv\u003eStatin \/ medication response (pharmacogenomics)\u003c\/div\u003e\n\u003cdiv class=\"x\" style=\"text-align: center;\"\u003eNever\u003c\/div\u003e\n\u003cdiv class=\"chk\" style=\"text-align: center;\"\u003eYes\u003c\/div\u003e\n\u003c\/div\u003e\n\u003cdiv class=\"nhd-compare-row\"\u003e\n\u003cdiv\u003eDetects silent risk before symptoms\u003c\/div\u003e\n\u003cdiv class=\"x\" style=\"text-align: center;\"\u003ePoorly\u003c\/div\u003e\n\u003cdiv class=\"chk\" style=\"text-align: center;\"\u003eComprehensively\u003c\/div\u003e\n\u003c\/div\u003e\n\u003c\/div\u003e\n\u003cdiv class=\"nhd-section\"\u003e\n\u003cdiv class=\"nhd-h3\"\u003eWhat about a stress test or coronary calcium scan?\u003c\/div\u003e\n\u003cp\u003eThese tests have their place — but they tell you what has already happened, not what's happening right now in your blood and vessel walls.\u003c\/p\u003e\n\u003cdiv class=\"nhd-warn\"\u003e\n\u003cstrong\u003eStress test:\u003c\/strong\u003e Detects blockages only after arteries are 70–80% narrowed. By that point, you're already deep into disease. A normal stress test does not mean you are not at risk — it means you haven't had enough damage yet for it to show up on the treadmill.\u003c\/div\u003e\n\u003cdiv class=\"nhd-warn\"\u003e\n\u003cstrong\u003eCoronary calcium scan (CAC):\u003c\/strong\u003e Measures calcified plaque that's already formed. A score of zero feels reassuring — but it misses soft, unstable plaque that hasn't calcified yet, which is often the type most likely to rupture and cause a heart attack. It also delivers radiation and gives no actionable biological information. You can't treat a calcium score. You can treat ApoB, inflammation, oxidative stress, and insulin resistance.\u003c\/div\u003e\n\u003cdiv class=\"nhd-warn\"\u003e\n\u003cstrong\u003eStandard lipid panel:\u003c\/strong\u003e The most dangerous test of all — not because it's harmful, but because it's falsely reassuring. Total cholesterol and LDL-C alone miss over half of cardiovascular risk. They don't measure particle size, particle number, oxidation state, inflammatory load, or genetic predisposition. \"Normal\" cholesterol kills people every day.\u003c\/div\u003e\n\u003cp\u003eThe Cardio Zoomer doesn't wait for disease to become visible. It reads the biological signals happening right now — in your blood, your vessels, and your DNA — while there's still time to change the outcome.\u003c\/p\u003e\n\u003c\/div\u003e\n\u003cdiv class=\"nhd-quote\"\u003e\"I've been practicing cardiology since 2002. I've sat with patients who passed every standard test with flying colors — and then had a heart attack six months later. Those patients deserved better. The Cardio Zoomer is the test I wish I'd had for them.\" \u003ccite\u003e— Dr. Jack Wolfson, DO, FACC — Natural Heart Doctor\u003c\/cite\u003e\n\u003c\/div\u003e\n\u003cdiv class=\"nhd-section\"\u003e\n\u003cdiv class=\"nhd-h3\"\u003eWhat we find — and what you can do about it\u003c\/div\u003e\n\u003cp\u003eEvery marker on the Cardio Zoomer has a meaning and an action. This isn't a report that sits in a drawer. It's a roadmap.\u003c\/p\u003e\n\u003c\/div\u003e\n\u003cdiv class=\"nhd-risk-block\"\u003e\n\u003cdiv class=\"nhd-risk-head\"\u003eApoB \u0026amp; Advanced Lipids — the real measure of plaque risk\u003c\/div\u003e\n\u003cdiv class=\"nhd-risk-body\"\u003e\n\u003cdiv\u003e\n\u003cdiv class=\"nhd-risk-col-label\"\u003eWhy it matters\u003c\/div\u003e\n\u003cp\u003eApoB counts the actual number of atherogenic particles in your blood — each one capable of burrowing into an artery wall. LDL-C measures their weight, not their count. You can have low LDL-C and dangerously high ApoB. Small dense LDL particles are 3–5x more likely to penetrate artery walls than large, buoyant particles — and a standard panel can't distinguish them.\u003c\/p\u003e\n\u003c\/div\u003e\n\u003cdiv\u003e\n\u003cdiv class=\"nhd-risk-col-label\"\u003eHow to improve it\u003c\/div\u003e\n\u003cp\u003eEliminate seed oils and processed carbohydrates. Prioritize wild seafood, pasture-raised meats, and healthy animal fats. Intermittent fasting lowers triglycerides and shifts LDL to large, buoyant particles. Resistance training improves lipid particle size. Optimize thyroid function — hypothyroidism is a leading driver of elevated ApoB.\u003c\/p\u003e\n\u003c\/div\u003e\n\u003c\/div\u003e\n\u003c\/div\u003e\n\u003cdiv class=\"nhd-risk-block\"\u003e\n\u003cdiv class=\"nhd-risk-head\"\u003eLp(a) — the inherited risk most doctors never check\u003c\/div\u003e\n\u003cdiv class=\"nhd-risk-body\"\u003e\n\u003cdiv\u003e\n\u003cdiv class=\"nhd-risk-col-label\"\u003eWhy it matters\u003c\/div\u003e\n\u003cp\u003eLp(a) is genetically determined and affects 1 in 5 people. It promotes clot formation and arterial inflammation independent of everything else. Elevated Lp(a) is one of the strongest independent predictors of heart attack and stroke — and it is almost never tested in standard care. You cannot know your true cardiovascular risk without it.\u003c\/p\u003e\n\u003c\/div\u003e\n\u003cdiv\u003e\n\u003cdiv class=\"nhd-risk-col-label\"\u003eHow to improve it\u003c\/div\u003e\n\u003cp\u003eLp(a) is largely genetic, but niacin (vitamin B3), estrogen therapy in postmenopausal women, and emerging RNA-based therapies can reduce levels. More importantly, knowing your Lp(a) lets you aggressively manage every other modifiable risk factor — which is exactly what we do at Natural Heart Doctor.\u003c\/p\u003e\n\u003c\/div\u003e\n\u003c\/div\u003e\n\u003c\/div\u003e\n\u003cdiv class=\"nhd-risk-block\"\u003e\n\u003cdiv class=\"nhd-risk-head\"\u003eInflammation (hsCRP, IL-6, TNF-α, Homocysteine) — the fire in your arteries\u003c\/div\u003e\n\u003cdiv class=\"nhd-risk-body\"\u003e\n\u003cdiv\u003e\n\u003cdiv class=\"nhd-risk-col-label\"\u003eWhy it matters\u003c\/div\u003e\n\u003cp\u003eChronic low-grade inflammation is the underlying driver of plaque formation, plaque rupture, and acute cardiac events. hsCRP is a stronger predictor of heart attack than LDL-C. IL-6 and TNF-α signal systemic inflammatory load. Elevated homocysteine damages the arterial lining directly and accelerates plaque development.\u003c\/p\u003e\n\u003c\/div\u003e\n\u003cdiv\u003e\n\u003cdiv class=\"nhd-risk-col-label\"\u003eHow to improve it\u003c\/div\u003e\n\u003cp\u003eRemove inflammatory foods: gluten, seed oils, processed sugar, and alcohol. Optimize omega-3 intake through wild salmon, sardines, and fish roe. Supplement magnesium, B vitamins (especially B6, B12, folate for homocysteine), and vitamin D. Prioritize 7–9 hours of sleep. Reduce chronic stress through movement, sunlight, and community.\u003c\/p\u003e\n\u003c\/div\u003e\n\u003c\/div\u003e\n\u003c\/div\u003e\n\u003cdiv class=\"nhd-risk-block\"\u003e\n\u003cdiv class=\"nhd-risk-head\"\u003eOxidative Stress (8-OHdG, F2-Isoprostane, oxLDL) — how fast you're rusting\u003c\/div\u003e\n\u003cdiv class=\"nhd-risk-body\"\u003e\n\u003cdiv\u003e\n\u003cdiv class=\"nhd-risk-col-label\"\u003eWhy it matters\u003c\/div\u003e\n\u003cp\u003eOxidative stress damages DNA, oxidizes LDL particles (making them toxic to arteries), and degrades the vessel lining. 8-OHdG measures DNA oxidative damage — elevated levels signal accelerated cellular aging and cancer risk alongside cardiovascular risk. F2-Isoprostanes are the gold standard marker of lipid peroxidation. Oxidized LDL is the true dangerous form of cholesterol.\u003c\/p\u003e\n\u003c\/div\u003e\n\u003cdiv\u003e\n\u003cdiv class=\"nhd-risk-col-label\"\u003eHow to improve it\u003c\/div\u003e\n\u003cp\u003eFlood your body with antioxidants from real food: wild berries, dark leafy greens, cacao, and colorful vegetables. Eliminate seed oils — they are highly susceptible to oxidation and the primary dietary driver of lipid peroxidation. Optimize glutathione through N-acetylcysteine, glycine, and selenium-rich foods like Brazil nuts and wild seafood.\u003c\/p\u003e\n\u003c\/div\u003e\n\u003c\/div\u003e\n\u003c\/div\u003e\n\u003cdiv class=\"nhd-risk-block\"\u003e\n\u003cdiv class=\"nhd-risk-head\"\u003eEndothelial Function (ADMA, SDMA, Nitric Oxide) — the health of your vessel walls\u003c\/div\u003e\n\u003cdiv class=\"nhd-risk-body\"\u003e\n\u003cdiv\u003e\n\u003cdiv class=\"nhd-risk-col-label\"\u003eWhy it matters\u003c\/div\u003e\n\u003cp\u003eThe endothelium — the one-cell-thin lining of your blood vessels — is your cardiovascular system's first and most critical line of defense. ADMA blocks nitric oxide production, causing vessels to stiffen, blood pressure to rise, and clotting risk to increase. Endothelial dysfunction precedes every other cardiovascular disease process and is present years before a diagnosis.\u003c\/p\u003e\n\u003c\/div\u003e\n\u003cdiv\u003e\n\u003cdiv class=\"nhd-risk-col-label\"\u003eHow to improve it\u003c\/div\u003e\n\u003cp\u003eSunlight is the most powerful nitric oxide booster available — UV exposure releases nitric oxide from skin stores into circulation. Dietary nitrates from beets, arugula, and leafy greens support production. L-arginine and L-citrulline (found in watermelon and supplementally) directly fuel the nitric oxide pathway. Daily movement and cold exposure also enhance endothelial function.\u003c\/p\u003e\n\u003c\/div\u003e\n\u003c\/div\u003e\n\u003c\/div\u003e\n\u003cdiv class=\"nhd-risk-block\"\u003e\n\u003cdiv class=\"nhd-risk-head\"\u003eInsulin Resistance \u0026amp; Metabolic Markers (HOMA-IR, TMAO, Uric Acid) — the hidden driver\u003c\/div\u003e\n\u003cdiv class=\"nhd-risk-body\"\u003e\n\u003cdiv\u003e\n\u003cdiv class=\"nhd-risk-col-label\"\u003eWhy it matters\u003c\/div\u003e\n\u003cp\u003eInsulin resistance is present in an estimated 88% of American adults to some degree — most don't know it. It drives triglyceride elevation, small dense LDL formation, hypertension, and systemic inflammation. TMAO (produced by gut bacteria from red meat and processed foods) accelerates atherosclerosis. Elevated uric acid stiffens arteries and raises blood pressure independently of gout.\u003c\/p\u003e\n\u003c\/div\u003e\n\u003cdiv\u003e\n\u003cdiv class=\"nhd-risk-col-label\"\u003eHow to improve it\u003c\/div\u003e\n\u003cp\u003eEliminate processed carbohydrates and sugar. Implement a time-restricted eating window (eating during daylight hours). Prioritize muscle-building exercise — muscle is the primary tissue for glucose disposal. Optimize gut health with fermented foods and diverse plant fibers to shift TMAO-producing bacterial populations. Reduce fructose, which is the primary driver of uric acid elevation.\u003c\/p\u003e\n\u003c\/div\u003e\n\u003c\/div\u003e\n\u003c\/div\u003e\n\u003cdiv class=\"nhd-risk-block\"\u003e\n\u003cdiv class=\"nhd-risk-head\"\u003eCardiovascular Genetics — the blueprint you were born with\u003c\/div\u003e\n\u003cdiv class=\"nhd-risk-body\"\u003e\n\u003cdiv\u003e\n\u003cdiv class=\"nhd-risk-col-label\"\u003eWhy it matters\u003c\/div\u003e\n\u003cp\u003eGenetics explain why two people with identical diets and lifestyles can have radically different cardiovascular outcomes. APOE4 carriers have significantly elevated Alzheimer's and cardiovascular risk. MTHFR variants impair folate metabolism and raise homocysteine. Factor V Leiden and prothrombin mutations elevate clotting risk. SLCO1B1 variants determine whether statins accumulate to toxic levels in muscle tissue.\u003c\/p\u003e\n\u003c\/div\u003e\n\u003cdiv\u003e\n\u003cdiv class=\"nhd-risk-col-label\"\u003eHow to improve it\u003c\/div\u003e\n\u003cp\u003eGenetics aren't destiny — they're a map. APOE4 carriers respond dramatically to dietary fat composition changes. MTHFR variants are addressed with methylated B vitamins. Clotting gene variants inform decisions around aspirin, omega-3 dosing, and lifestyle modifications. And knowing your statin pharmacogenomics could prevent serious muscle damage if medication is ever considered.\u003c\/p\u003e\n\u003c\/div\u003e\n\u003c\/div\u003e\n\u003c\/div\u003e\n\u003cdiv class=\"nhd-section\"\u003e\n\u003cdiv class=\"nhd-h3\"\u003eHow it works\u003c\/div\u003e\n\u003cdiv class=\"nhd-steps\"\u003e\n\u003cdiv class=\"nhd-step\"\u003e\n\u003cdiv class=\"nhd-step-num\"\u003e1\u003c\/div\u003e\n\u003cdiv class=\"nhd-step-title\"\u003eOrder\u003c\/div\u003e\n\u003cdiv class=\"nhd-step-body\"\u003eDr. Wolfson orders the test for you as your provider\u003c\/div\u003e\n\u003c\/div\u003e\n\u003cdiv class=\"nhd-step\"\u003e\n\u003cdiv class=\"nhd-step-num\"\u003e2\u003c\/div\u003e\n\u003cdiv class=\"nhd-step-title\"\u003eCollect\u003c\/div\u003e\n\u003cdiv class=\"nhd-step-body\"\u003eBlood draw at a local lab + simple home urine sample\u003c\/div\u003e\n\u003c\/div\u003e\n\u003cdiv class=\"nhd-step\"\u003e\n\u003cdiv class=\"nhd-step-num\"\u003e3\u003c\/div\u003e\n\u003cdiv class=\"nhd-step-title\"\u003eReturn\u003c\/div\u003e\n\u003cdiv class=\"nhd-step-body\"\u003eShip samples with the prepaid return label in your kit\u003c\/div\u003e\n\u003c\/div\u003e\n\u003cdiv class=\"nhd-step\"\u003e\n\u003cdiv class=\"nhd-step-num\"\u003e4\u003c\/div\u003e\n\u003cdiv class=\"nhd-step-title\"\u003eResults\u003c\/div\u003e\n\u003cdiv class=\"nhd-step-body\"\u003eFull report delivered to your provider in ~10 business days\u003c\/div\u003e\n\u003c\/div\u003e\n\u003c\/div\u003e\n\u003c\/div\u003e\n\u003cdiv class=\"nhd-who\"\u003e\n\u003cspan class=\"nhd-who-label\"\u003eThis test is for you if:\u003c\/span\u003e\n\u003cul\u003e\n\u003cli\u003eYou've been told your cholesterol is \"fine\" — but you're not convinced\u003c\/li\u003e\n\u003cli\u003eYou have a family history of heart attack or stroke, especially before age 65\u003c\/li\u003e\n\u003cli\u003eYou want to know your real cardiovascular risk — not a guess based on age and LDL\u003c\/li\u003e\n\u003cli\u003eYou've already had a cardiac event and want to understand the root cause\u003c\/li\u003e\n\u003cli\u003eYou're on or considering a statin and want to understand your pharmacogenomic response\u003c\/li\u003e\n\u003cli\u003eYou have high blood pressure, diabetes, or metabolic syndrome\u003c\/li\u003e\n\u003cli\u003eYou're committed to living to 100 with a strong heart — and you want the data to get there\u003c\/li\u003e\n\u003c\/ul\u003e\n\u003c\/div\u003e\n\u003cdiv class=\"nhd-section\"\u003e\n\u003cdiv class=\"nhd-markers-head\"\u003e\n\u003cdiv class=\"nhd-h3\"\u003eFull marker list\u003c\/div\u003e\n\u003cspan class=\"nhd-badge\"\u003e200+ total markers\u003c\/span\u003e\n\u003c\/div\u003e\n\u003cdiv class=\"nhd-row\"\u003e\n\u003cspan class=\"nhd-row-label\"\u003eRisk Scores\u003c\/span\u003e\u003cspan class=\"nhd-row-count\"\u003e2\u003c\/span\u003e\u003cspan class=\"nhd-row-markers\"\u003eReynolds Risk Score, Framingham Risk Score\u003c\/span\u003e\n\u003c\/div\u003e\n\u003cdiv class=\"nhd-row\"\u003e\n\u003cspan class=\"nhd-row-label\"\u003eMetabolic Risk Markers\u003c\/span\u003e\u003cspan class=\"nhd-row-count\"\u003e30\u003c\/span\u003e\u003cspan class=\"nhd-row-markers\"\u003eHbA1c, Glucose, Glycated Serum Protein, Insulin, C-Peptide, HOMA-IR, Adiponectin, TMAO, L-Carnitine, Ferritin, Leptin, Sodium, Potassium, Chloride, CO2, BUN, Creatinine, eGFR, BUN\/Creatinine Ratio, Serum Osmolality, Uric Acid, Cystatin C, ALT, AST, GGT, Bilirubin, Total Protein, Alkaline Phosphatase, Glucose (Renal)\u003c\/span\u003e\n\u003c\/div\u003e\n\u003cdiv class=\"nhd-row\"\u003e\n\u003cspan class=\"nhd-row-label\"\u003eLipids \u0026amp; Ratios\u003c\/span\u003e\u003cspan class=\"nhd-row-count\"\u003e15\u003c\/span\u003e\u003cspan class=\"nhd-row-markers\"\u003eTotal Cholesterol, Triglycerides, HDL Direct, Non-HDL-C, LDL Direct, Small Dense LDL, Lp(a), ApoA-1, ApoB, TC\/HDL, TG\/VLDL, ApoB\/ApoA-1, HDL\/TG\u003c\/span\u003e\n\u003c\/div\u003e\n\u003cdiv class=\"nhd-row\"\u003e\n\u003cspan class=\"nhd-row-label\"\u003eCeramides \u0026amp; Ratios\u003c\/span\u003e\u003cspan class=\"nhd-row-count\"\u003e6\u003c\/span\u003e\u003cspan class=\"nhd-row-markers\"\u003eCer(d18:1\/16:0), Cer(d18:1\/18:0), Cer(d18:1\/24:1) and their ratios vs Cer(d18:1\/24:0)\u003c\/span\u003e\n\u003c\/div\u003e\n\u003cdiv class=\"nhd-row\"\u003e\n\u003cspan class=\"nhd-row-label\"\u003eSterols\u003c\/span\u003e\u003cspan class=\"nhd-row-count\"\u003e5\u003c\/span\u003e\u003cspan class=\"nhd-row-markers\"\u003eSterol Balance Score, Desmosterol, Lathosterol, Beta-Sitosterol, Campesterol\u003c\/span\u003e\n\u003c\/div\u003e\n\u003cdiv class=\"nhd-row\"\u003e\n\u003cspan class=\"nhd-row-label\"\u003eOxidative Stress (Redox)\u003c\/span\u003e\u003cspan class=\"nhd-row-count\"\u003e5\u003c\/span\u003e\u003cspan class=\"nhd-row-markers\"\u003e8-OHdG, F2-Isoprostane, Malondialdehyde, Nitrotyrosine, Chlorotyrosine\u003c\/span\u003e\n\u003c\/div\u003e\n\u003cdiv class=\"nhd-row\"\u003e\n\u003cspan class=\"nhd-row-label\"\u003eOmega Fatty Acids\u003c\/span\u003e\u003cspan class=\"nhd-row-count\"\u003e7\u003c\/span\u003e\u003cspan class=\"nhd-row-markers\"\u003eEPA, DPA, DHA, Linoleic Acid (LA), Arachidonic Acid (AA), AA\/EPA Ratio, Omega-3 Index\u003c\/span\u003e\n\u003c\/div\u003e\n\u003cdiv class=\"nhd-row\"\u003e\n\u003cspan class=\"nhd-row-label\"\u003eInflammation Markers\u003c\/span\u003e\u003cspan class=\"nhd-row-count\"\u003e6\u003c\/span\u003e\u003cspan class=\"nhd-row-markers\"\u003ehsCRP, Homocysteine, IL-6, TNF-α, Omega-6 Total, Omega-3 Total\u003c\/span\u003e\n\u003c\/div\u003e\n\u003cdiv class=\"nhd-row\"\u003e\n\u003cspan class=\"nhd-row-label\"\u003eEndothelial Dysfunction\u003c\/span\u003e\u003cspan class=\"nhd-row-count\"\u003e8\u003c\/span\u003e\u003cspan class=\"nhd-row-markers\"\u003eADMA, SDMA, Homoarginine, Arginine, Citrulline, Arginine\/ADMA Ratio, Arginine\/SDMA Ratio, Choline\u003c\/span\u003e\n\u003c\/div\u003e\n\u003cdiv class=\"nhd-row\"\u003e\n\u003cspan class=\"nhd-row-label\"\u003eMacrophage \u0026amp; Plaque\u003c\/span\u003e\u003cspan class=\"nhd-row-count\"\u003e3\u003c\/span\u003e\u003cspan class=\"nhd-row-markers\"\u003eMyeloperoxidase (MPO), PLAC, Oxidized LDL (oxLDL)\u003c\/span\u003e\n\u003c\/div\u003e\n\u003cdiv class=\"nhd-row\"\u003e\n\u003cspan class=\"nhd-row-label\"\u003eCardiac Stress \u0026amp; Clotting\u003c\/span\u003e\u003cspan class=\"nhd-row-count\"\u003e3\u003c\/span\u003e\u003cspan class=\"nhd-row-markers\"\u003eNT-proBNP, Troponin-T, Creatine Kinase\u003c\/span\u003e\n\u003c\/div\u003e\n\u003cdiv class=\"nhd-row\"\u003e\n\u003cspan class=\"nhd-row-label\"\u003eLipid Metabolism Genes\u003c\/span\u003e\u003cspan class=\"nhd-row-count\"\u003e30+\u003c\/span\u003e\u003cspan class=\"nhd-row-markers\"\u003eAPOE, APOB, APOA1, APOA2, APOC3, APOA5, LDLR, LDLRAP1, PCSK9, LPL, CETP, LCAT, SCARB1, ANGPTL3, ANGPTL4, MTP, SORT1, TCF7L2, GCKR, SCD1, CYP27A1, and more\u003c\/span\u003e\n\u003c\/div\u003e\n\u003cdiv class=\"nhd-row\"\u003e\n\u003cspan class=\"nhd-row-label\"\u003eVascular \u0026amp; BP Genes\u003c\/span\u003e\u003cspan class=\"nhd-row-count\"\u003e30+\u003c\/span\u003e\u003cspan class=\"nhd-row-markers\"\u003eNOS1, NOS2, NOS3, ACE, AGT, AGTR1, MTHFR, COMT, EDN1, ADD1, CORIN, CYP4A11, CYP4F2, CYP11B2, VEGF-A, 9p21 region, 4q25, CD36, and more\u003c\/span\u003e\n\u003c\/div\u003e\n\u003cdiv class=\"nhd-row\"\u003e\n\u003cspan class=\"nhd-row-label\"\u003ePharmacogenomic Variants\u003c\/span\u003e\u003cspan class=\"nhd-row-count\"\u003e19\u003c\/span\u003e\u003cspan class=\"nhd-row-markers\"\u003eSLCO1B1 (statin transport), CYP2C9, CYP2C19, CYP3A5, CYP1A2, KIF6, BCRP\/ABCG2, OCT2, COX-1, NQO1, HO1, AT1R, BDKRB2, PNPLA3, DGAT2\u003c\/span\u003e\n\u003c\/div\u003e\n\u003cdiv class=\"nhd-row\"\u003e\n\u003cspan class=\"nhd-row-label\"\u003eInflammation \u0026amp; Oxidative Genes\u003c\/span\u003e\u003cspan class=\"nhd-row-count\"\u003e8\u003c\/span\u003e\u003cspan class=\"nhd-row-markers\"\u003ePON1, GPX1, MPO, ILR-6, LIPA, ALDH2, HDAC9, CDKN2B-AS1\u003c\/span\u003e\n\u003c\/div\u003e\n\u003cdiv class=\"nhd-row\"\u003e\n\u003cspan class=\"nhd-row-label\"\u003eClotting \u0026amp; Thrombosis Genes\u003c\/span\u003e\u003cspan class=\"nhd-row-count\"\u003e4\u003c\/span\u003e\u003cspan class=\"nhd-row-markers\"\u003eFactor V Leiden (rs6025), Prothrombin (rs1799963, rs3136516), PLG (rs4252120)\u003c\/span\u003e\n\u003c\/div\u003e\n\u003c\/div\u003e\n\u003cdiv class=\"nhd-cta\"\u003e\n\u003ch3\u003eThe test that could save your life is right here.\u003c\/h3\u003e\n\u003cp\u003eHeart disease is preventable. It is reversible. But only if you know what you're dealing with. Order the Cardio Zoomer today and get the most complete cardiovascular risk picture available anywhere — then let Dr. Wolfson and the Natural Heart Doctor team show you exactly what to do with it.\u003c\/p\u003e\n\u003c\/div\u003e\n\u003cp class=\"nhd-disclaimer\"\u003e\u003cstrong\u003eImportant:\u003c\/strong\u003e The Cardio Zoomer is a clinical laboratory test ordered by a licensed healthcare provider. Results are intended to inform lifestyle, nutrition, and clinical decisions — not to diagnose disease. This test is not available in New York State. Lab processing by Vibrant Wellness (CLIA-certified, CAP-accredited facility). Results available in approximately 10 business days after sample receipt. HSA\/FSA eligible. No insurance accepted.\u003c\/p\u003e\n\u003c\/div\u003e","brand":"Natural Heart Doctor","offers":[{"title":"Default Title","offer_id":49492574437616,"sku":null,"price":1175.0,"currency_code":"USD","in_stock":true}],"thumbnail_url":"\/\/cdn.shopify.com\/s\/files\/1\/0609\/0470\/2192\/files\/cardio_zoomer_plus_genetics.png?v=1775777145","url":"https:\/\/shop.naturalheartdoctor.com\/products\/cardio-zoomer-plus-cardio-genetics","provider":"Natural Heart Doctor Shop","version":"1.0","type":"link"}